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AR cooperates with SMAD4 to maintain skeletal muscle homeostasis.

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Forouhan M. et al, (2022), Acta Neuropathol

Nuclear microRNA-466c regulates Vegfa expression in response to hypoxia

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Laitinen P. et al, (2022), PLoS ONE, 17

Enhancing the Therapeutic Potential of Extracellular Vesicles Using Peptide Technology

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Martin Perez C. et al, (2022), Methods in Molecular Biology, 2383, 119 - 141

Molecular correction of Duchenne muscular dystrophy by splice modulation and gene editing.

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Hanson B. et al, (2021), RNA Biol, 1 - 15

Application of CRISPR-Cas9-Mediated Genome Editing for the Treatment of Myotonic Dystrophy Type 1.

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Marsh S. et al, (2020), Mol Ther, 28, 2527 - 2539

Mutation-independent Proteomic Signatures of Pathological Progression in Murine Models of Duchenne Muscular Dystrophy.

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van Westering TLE. et al, (2020), Mol Cell Proteomics, 19, 2047 - 2068

Advances in oligonucleotide drug delivery.

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Roberts TC. et al, (2020), Nat Rev Drug Discov, 19, 673 - 694

Uniform sarcolemmal dystrophin expression is required to prevent extracellular microRNA release and improve dystrophic pathology.

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van Westering TLE. et al, (2020), J Cachexia Sarcopenia Muscle, 11, 578 - 593

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