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In mice, the pathway involving PIWI and PIWI-interacting RNA (PIWI-piRNA) is essential to re-establish transposon silencing during male-germline reprogramming. The cytoplasmic PIWI protein MILI mediates piRNA-guided transposon RNA cleavage as well as piRNA amplification. MIWI2's binding to piRNA and its nuclear localization are proposed to be dependent upon MILI function. Here, we demonstrate the existence of a piRNA biogenesis pathway that sustains partial MIWI2 function and reprogramming activity in the absence of MILI.

Original publication

DOI

10.1038/nsmb.3413

Type

Journal article

Journal

Nat Struct Mol Biol

Publication Date

07/2017

Volume

24

Pages

604 - 606

Keywords

Animals, Argonaute Proteins, Gene Expression Regulation, Mice, Mice, Knockout, RNA, Small Interfering