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\n Author Correction: Sex-specific innate immune selection of HIV-1 in utero is associated with increased female susceptibility to infection.\n

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Adland E. et al, (2020), Nat Commun, 11

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\n HIGH-FREQUENCY failure of combination antiretroviral therapy in paediatric HIV infection is associated with unmet maternal needs causing maternal NON-ADHERENCE.\n

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Millar JR. et al, (2020), EClinicalMedicine, 22

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\n Sex-specific innate immune selection of HIV-1 in utero is associated with increased female susceptibility to infection.\n

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Adland E. et al, (2020), Nat Commun, 11

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\n In Reply: Insufficient Evidence to Diagnose In Utero Infection of HIV.\n

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Millar J. and Goulder P., (2020), Pediatr Infect Dis J, 39, e11 - e12

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\n Erratum: Elevated HLA-A expression impairs HIV control through inhibition of NKG2A-expressing cells (Science DOI: 10.1126/science.aam8825)\n

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Ramsuran V. et al, (2019), Science, 365

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\n Increasing Diagnostic Uncertainties in Children With In Utero HIV Infection.\n

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Millar JR. et al, (2019), Pediatr Infect Dis J

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\n Differential Pathogen-Specific Immune Reconstitution in Antiretroviral Therapy-Treated Human Immunodeficiency Virus-Infected Children.\n

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Muenchhoff M. et al, (2019), J Infect Dis, 219, 1407 - 1417

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\n Impact of HLA Allele-KIR Pairs on HIV Clinical Outcome in South Africa.\n

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Mori M. et al, (2019), J Infect Dis, 219, 1456 - 1463

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\n Recommendations for analytical antiretroviral treatment interruptions in HIV research trials-report of a consensus meeting.\n

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Julg B. et al, (2019), Lancet HIV, 6, e259 - e268

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\n HBV vaccination and PMTCT as elimination tools in the presence of HIV: insights from a clinical cohort and dynamic model.\n

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McNaughton AL. et al, (2019), BMC Med, 17

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\n Virological remission after antiretroviral therapy interruption in female African HIV seroconverters.\n

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Gossez M. et al, (2019), AIDS, 33, 185 - 197

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\n Strong sex bias in elite control of paediatric HIV infection.\n

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Vieira VA. et al, (2019), AIDS, 33, 67 - 75

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\n Increased Regulatory T-Cell Activity and Enhanced T-Cell Homeostatic Signaling in Slow Progressing HIV-infected Children.\n

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Roider J. et al, (2019), Front Immunol, 10

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\n Plasma IL-5 but not CXCL13 correlates with neutralization breadth in HIV-infected children\n

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Roider J. et al, (2019), Frontiers in Immunology, 10

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\n HIV control: Is getting there the same as staying there?\n

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Goulder P. and Deeks SG., (2018), PLoS Pathog, 14

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\n Broadly Neutralizing Antibodies in Children Correlate With Increased HIV-specific T-follicular Helper and Regulatory Cells Within Germinal Centers\n

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Roider J. et al, (2018), AIDS RESEARCH AND HUMAN RETROVIRUSES, 34, 75 - 75

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\n HIV-1 Subtype C-Infected Children with Exceptional Neutralization Breadth Exhibit Polyclonal Responses Targeting Known Epitopes.\n

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Ditse Z. et al, (2018), J Virol, 92

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\n Unmet needs of high-risk mothers reduce success of antiretroviral treatment in HIV-infected infants.\n

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Mvo Z. et al, (2018), S Afr Med J, 108, 609 - 610

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\n Recovery of effective HIV-specific CD4+ T-cell activity following antiretroviral therapy in paediatric infection requires sustained suppression of viraemia.\n

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Adland E. et al, (2018), AIDS, 32, 1413 - 1422

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\n Frequent generation of HIV broadly neutralizing antibodies in infected children is associated with both increased help and regulation within germinal centers\n

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Roider J. et al, (2018), JOURNAL OF THE INTERNATIONAL AIDS SOCIETY, 21, 32 - 32

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